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2 edition of Clinical determinants of weight loss in obese humans found in the catalog.

Clinical determinants of weight loss in obese humans

Arnoud C. Toornvliet

Clinical determinants of weight loss in obese humans

pharmacological and nutritional methods

by Arnoud C. Toornvliet

  • 306 Want to read
  • 18 Currently reading

Published by University of Leiden] in [Leiden .
Written in English


Edition Notes

StatementArnoud C. Toornvliet.
Classifications
LC ClassificationsMLCM 2002/02514
The Physical Object
Pagination136 p. :
Number of Pages136
ID Numbers
Open LibraryOL478426M
ISBN 109090113843
LC Control Number98204558

  Weight loss on a low-fat diet: consequence of the imprecision of the control of food intake in humans. American Journal of Clinical Nutrition ; 53 . Obesity is currently regarded as one of the major health challenges of the developed world. Excess body weight is an important risk factor for morbidity and .

  Another study with participants published in the July/August issue of Obesity Research & Clinical Practice found alternate day fasting my be more effective for middle aged people (50 to 59 years of age), as these participants had a greater weight loss during an eight-week alternate day fasting trial. Maintenance of weight loss Sub questions Review limitations Section 5 a: Management of obesity 2 15 Management of obesity in clinical s ettings General introduction to clinical management Children Factors to be considered in the clinical assessment of.

OBJECTIVE To examine the mechanisms by which weight loss improves glycemic control in overweight subjects with NIDDM, particularly the relationships between energy restriction, improvement in insulin sensitivity, andregional and overall adipose tissue loss. RESEARCH DESIGN AND METHODS Hyperinsulinemic glucose clamps wereperformed in 20 subjects .   There are many metabolic, hormonal, and clinical parameters that are altered in morbid obesity and can benefit from weight loss. However, for some of those (e.g., leptin levels), the observed variation after weight reduction is greater than expected 2, 3, whereas for others (e.g., blood pressure) the improvement is smaller than could be.


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Clinical determinants of weight loss in obese humans by Arnoud C. Toornvliet Download PDF EPUB FB2

It was found that dietary counseling, TNM stage, total energy intake, depression, esophagitis, and loss of appetite were the most important factors for weight loss.

The results underline the importance of maintaining energy intake and providing Cited by:   Thus, an increased understanding and the clinical characterization of phenotypic differences in energy metabolism among individuals (metabolic profile) may lead to new strategies to prevent weight gain or improve weight‐loss interventions by targeted therapies on the basis of metabolic phenotype and susceptibility to obesity in individual Cited by: Nutrients, foods, and beverages.

That dietary fat was a main cause of obesity was once a widespread belief. However, in an 8-year follow-up of NHS, total fat intake was only weakly related to weight gain.7 Increasing intakes of energy from monounsaturated or polyunsaturated fats were not associated with weight gain, whereas increasing energy intakes from animal, Cited by:   A total of subjects were included in the analysis.

The degree of weight loss achieved by subjects of varying age ranges is portrayed in Fig. 1A and B. Results reveal that subjects aged 50– y achieved greater (P = ) absolute and percent weight loss than subjects in other age groups. As for sex differences, there were no differences in the degree of weight loss Cited by: 8.

The dominant ideology of weight is that obesity is a disease, fat is bad, weight loss will improve health, and that weight loss is within personal control [, 20, 23]. and resultant obesity occur. The human body naturally resists and defends attempted weight loss (Fig.

1), with counter-regulatory responses driving hunger, for example, by reducing levels of hunger-suppressing hormones. In addition, there is slowing of the metabolic rate and a reduction in non-purposeful move. A team led by Dr.

Christopher D. Gardner of Stanford University explored the influence of certain biological factors on the success of weight loss with low-fat and low-carbohydrate diets. They analyzed the patterns of genetic variation (genotypes) in three genes that code for proteins involved in fat and carbohydrate metabolism.

Obese individuals also perceive that these public health messages a) overemphasise the physical health risks associated with obesity while underemphasising the social (e.g., stigma) and psychological (e.g., distress) dimensions, b) focus too much on weight measurement and weight loss, c) neglect discussion of the day-to-day management of.

Spreadsheet d. Weight Loss and Hypertension Risk: Lifestyles Trials, All Subjects. C–18 Spreadsheet e. Weight Loss and Hypertension Risk: Lifestyles Trials, Diabetes Subjects C–19 Spreadsheet f.

Weight Loss and Hypertension Risk: Lifestyles Trials, Hypertension. With success, and if warranted, further weight loss can be attempted. Weight loss should be about 1 to 2 pounds per week for a period of 6 months, with the subsequent strategy based on the amount of weight lost.

Low-calorie diets (LCD) for weight loss in overweight and obese persons. Similarly, a failure to increase fat oxidation during 1 day of % overfeeding of a diet with 60% calories from fats also predicted weight gain over the period of a year, indicating that impaired metabolic flexibility (or inflexibility) in the context of a high‐fat overfeeding diet is a metabolic predictor of weight gain.

Although the use. Clinical trials funded by the NIH or other government agencies focused on treating or managing overweight and obesity that are currently open and recruiting can be viewed at This is a curated list of clinical trials, but you can expand or narrow your search to find more clinical trials for overweight and obesity.

Some researchers have suggested that excess weight is protective against mortality, but this “obesity paradox” is likely observed because of confounding by smoking and existing or preclinical conditions that lead to weight loss preceding death (i.e., reverse causation).

41 When these methodological issues are correctly accounted for, as in. References: Effects of Moderate and Subsequent Progressive Weight Loss on Metabolic Function and Adipose Tissue Biology in Humans with Obesity.

Magkos F, Fraterrigo G, Yoshino J, Luecking C, Kirbach K, Kelly SC, de Las Fuentes L, He S, Okunade AL, Patterson BW, Klein S.

Cell Metab. Feb pii: S(16) doi: /   Another novel finding is that the determinants of weight-loss targets, in particular of realistic weight-loss “goal”, vary with individuals' BMI category. The most prominent result is related to the severe and morbid obesity categories. People included in these categories are at high risk of unrealistic weight-loss goal.

Consistently, weight loss has led to increased T levels in obese men [14, 15]. Although the underlying mechanisms are still unclear, these findings suggest that hypertrophy of the adipocytes and its related metabolic changes may associate with the decline in T levels in obese.

Objective: To explore the correlation models between body mass index (BMI) and sex hormones constructed from a male cross-sectional survey and evaluate the effects of surgery-induced weight loss on sex hormones in morbidly obese subjects that are not predicted by the constructed BMI correlation models.

Design: Cross-sectional population and longitudinal studies. AT was independent of macronutrient composition of the diet [44–47].It was thought to be proportional to the degree of weight loss with an additional effect of baseline weight [].However, analysing data on obese patients before and after weight loss [1, 55, 56], the association between weight change and AT was weak (see Fig.

2).This was in line with the results of a controlled clinical. Results After adjustment for BMI and duration, there was no significant difference in mean body weight loss (VLCD kg more weight loss than ADF, 95% CI: −, ) or fat.

Obesity means having excess body fat. Adults 35 years of age and older with a BMI greater than 30 are obese.; Obesity is not just a cosmetic consideration. It is a chronic medical disease that can lead to diabetes, high blood pressure, obesity associated cardiovascular disease such as heart disease, gallstones, and other chronic illnesses.; Obesity is a risk factor for a number of.

Lifestyle interventions leading to weight loss generally induce preferential mobilization of visceral fat. In clinical practice, measuring waist circumference in addition to the body mass index could be helpful for the identification and management of a subgroup of overweight or obese patients at high cardiometabolic risk.Ideology, obesity and the social determinants of health: a critical analysis of the obesity and health relationship Based on a critical review of the obesity and health literature we provide five models of how the hypothesized obesity and health relationship is conceptualized.JAMY D.

ARD MD, in Handbook of Clinical Nutrition (Fourth Edition), Complications Associated with Increased Intra-abdominal Fat. Body fat distribution is a better predictor of the health hazards of obesity than is the absolute amount of body fat.

Individuals with an upper body fat pattern, reflecting an excess of intra-abdominal or visceral fat, have significantly greater .